Erratum: Cardiac RKIP induces a beneficial β-adrenoceptor–dependent positive inotropy
نویسندگان
چکیده
منابع مشابه
Cardiac Glycoside Receptors and Positive Inotropy
With Contributions by L .Brown, W.H.Barry , D.Charlemagne, E. Erdmann, A . A . L.Fox, U.Fricke, W.H.van Gilst, T.Godfraind, K.Greeff, M.Heller , Κ.-Ο. Haustein, G. Isenberg, R.Jacob, T.Kazazoglou, D . K i m , K.Kochsiek, J .M.Koomen, W.Krawietz, M.Lazdunski, L.G.Lelievre, H.Lül lmann, P.Mansicr, E.Noack, D.Noble, D.Palm, Th.Peters, H.-H.Pri l lwitz , J.F.Renaud, B.Rossi, J . A . M . Schevers, T...
متن کاملCardiac α1-Adrenoceptors and Inotropy
Cardiac α1-adrenoceptor stimulation elicits a positive inotropic effect because of myofilament Ca2+ sensitization with a small increase in Ca2+ transients predominantly mediated by α1B-adrenoceptors via the intracellular alkalinization and potential myosin light chain 2 phosphorylation. However, the α1-adrenoceptor– mediated inotropy exhibits a wide range of species-dependent variation. In addi...
متن کاملCardiac α1-Adrenoceptors and Inotropy
Cardiac α1-adrenoceptor stimulation elicits a positive inotropic effect because of myofilament Ca2+ sensitization with a small increase in Ca2+ transients predominantly mediated by α1B-adrenoceptors via the intracellular alkalinization and potential myosin light chain 2 phosphorylation. However, the α1-adrenoceptor– mediated inotropy exhibits a wide range of species-dependent variation. In addi...
متن کاملCardiac α1-Adrenoceptors and Inotropy
Cardiac α1-adrenoceptor stimulation elicits a positive inotropic effect because of myofilament Ca2+ sensitization with a small increase in Ca2+ transients predominantly mediated by α1B-adrenoceptors via the intracellular alkalinization and potential myosin light chain 2 phosphorylation. However, the α1-adrenoceptor– mediated inotropy exhibits a wide range of species-dependent variation. In addi...
متن کاملTotal β-Adrenoceptor Deficiency Results in Cardiac Hypotrophy and Negative Inotropy
S. LEE, S. GRAFWEG, T. SCHNEIDER, M. JIMENEZ, J.-P. GIACOBINO, A. GHANEM, K. TIEMANN, W. BLOCH, J. MÜLLER-EHMSEN, R. H. G. SCHWINGER, K. BRIXIUS Laboratory of Muscle Research and Molecular Cardiology, Department III of Internal Medicine, University of Cologne, Cologne, Germany, Institute of Neurophysiology, University of Cologne, Cologne, Germany, Départment of Medical Biochemistry, Medical Cen...
متن کاملذخیره در منابع من
با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید
ژورنال
عنوان ژورنال: Nature Medicine
سال: 2016
ISSN: 1078-8956,1546-170X
DOI: 10.1038/nm0216-217c